INT287598

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Context Info
Confidence 0.00
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.83
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

molecular_function (Dnajc13) cellular_component (Dnajc13) biological_process (Dnajc13)
Dnajc13 (Mus musculus)
Pain Link Frequency Relevance Heat
agonist 7 5.00 Very Low Very Low Very Low
Glutamate receptor 2 5.00 Very Low Very Low Very Low
Hippocampus 2 5.00 Very Low Very Low Very Low
depression 1 5.00 Very Low Very Low Very Low
Pain 1 5.00 Very Low Very Low Very Low
nMDA receptor antagonist 1 5.00 Very Low Very Low Very Low
alcohol 1 5.00 Very Low Very Low Very Low
Immobilon 1 5.00 Very Low Very Low Very Low
isoflurane 1 5.00 Very Low Very Low Very Low
imagery 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Fragile X Syndrome 21 97.12 Very High Very High Very High
Disease 9 80.32 Quite High
Autism 4 75.44 Quite High
Intellectual Impairment 9 74.56 Quite High
Liver Disease 5 74.16 Quite High
Syndrome 3 40.24 Quite Low
Congenital Anomalies 3 37.84 Quite Low
Targeted Disruption 9 5.00 Very Low Very Low Very Low
Li-fraumeni Syndrome 5 5.00 Very Low Very Low Very Low
Anxiety Disorder 5 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
These data suggest that loss of FMRP function, particularly in KH2-mediated RNA binding and in synaptic plasticity, play critical roles in pathogenesis of the Fragile X Syndrome and establish a new model for studying the disorder.


KH2-mediated Binding (binding) of associated with fragile x syndrome
1) Confidence 0.00 Published 2009 Journal PLoS Genetics Section Abstract Doc Link PMC2779495 Disease Relevance 0.83 Pain Relevance 0

General Comments

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