INT288189

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Context Info
Confidence 0.26
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.43
Pain Relevance 0.28

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (Tlr2) plasma membrane (Tlr2) intracellular (Tlr2)
cytoplasm (Tlr2)
Tlr2 (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammatory response 13 97.84 Very High Very High Very High
Inflammation 27 86.88 High High
tolerance 14 81.52 Quite High
cytokine 38 65.44 Quite High
chemokine 2 5.00 Very Low Very Low Very Low
Arthritis 2 5.00 Very Low Very Low Very Low
addiction 1 5.00 Very Low Very Low Very Low
agonist 1 5.00 Very Low Very Low Very Low
cINOD 1 5.00 Very Low Very Low Very Low
antagonist 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
INFLAMMATION 41 97.66 Very High Very High Very High
Lyme Disease 13 96.98 Very High Very High Very High
Syndrome 1 44.56 Quite Low
Disease 4 25.00 Low Low
Pressure And Volume Under Development 4 5.00 Very Low Very Low Very Low
Infection 4 5.00 Very Low Very Low Very Low
Arthritis 2 5.00 Very Low Very Low Very Low
Disease Susceptibility 2 5.00 Very Low Very Low Very Low
Bacterial Infection 2 5.00 Very Low Very Low Very Low
Mycobacterial Infection 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Herein, this paradigm is challenged by demonstrating that recognition of live Borrelia burgdorferi not only triggers an inflammatory response in the absence of CD14, but one that is, in part, a consequence of altered PI3K/AKT/p38-MAPK signaling and impaired negative regulation of TLR2.
Negative_regulation (impaired) of Negative_regulation (regulation) of TLR2 associated with lyme disease and inflammatory response
1) Confidence 0.26 Published 2009 Journal PLoS Pathogens Section Abstract Doc Link PMC2781632 Disease Relevance 0.43 Pain Relevance 0.28

General Comments

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