INT288225

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Context Info
Confidence 0.26
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.58
Pain Relevance 0.08

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

AIS (Homo sapiens)
Pain Link Frequency Relevance Heat
Central nervous system 15 85.00 Quite High
nMDA receptor 1 40.88 Quite Low
cytokine 5 5.00 Very Low Very Low Very Low
Spinal cord 4 5.00 Very Low Very Low Very Low
Somatostatin 4 5.00 Very Low Very Low Very Low
addiction 3 5.00 Very Low Very Low Very Low
Mechanotransduction 1 5.00 Very Low Very Low Very Low
peripheral neuropathy 1 5.00 Very Low Very Low Very Low
Catecholamine 1 5.00 Very Low Very Low Very Low
antagonist 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Scoliosis 314 100.00 Very High Very High Very High
Stroke 2 92.88 High High
Congenital Anomalies 14 68.44 Quite High
Obesity 152 48.20 Quite Low
Targeted Disruption 6 22.32 Low Low
Stress 16 9.92 Low Low
Syndrome 16 5.00 Very Low Very Low Very Low
Osteoporosis 13 5.00 Very Low Very Low Very Low
Diabetes Mellitus 6 5.00 Very Low Very Low Very Low
Handedness 5 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The fate of early AIS - to progress, become static or resolve (rarely) according to the double neuro-osseous theory generally depends on the relative contribution and outcome of the disharmony (Figure 1) between:
Protein_catabolism (progress) of AIS associated with scoliosis
1) Confidence 0.26 Published 2009 Journal Scoliosis Section Body Doc Link PMC2781798 Disease Relevance 0.58 Pain Relevance 0.08

General Comments

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