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Context Info
Confidence 0.08
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.69
Pain Relevance 0.11

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (EPO, Nos3) mitochondrion (Nos3) aging (EPO)
Golgi apparatus (Nos3) cytoplasm (Nos3) extracellular space (EPO)
Anatomy Link Frequency
blood 1
EPO (Homo sapiens)
Nos3 (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammation 1 93.60 High High
Bioavailability 2 79.28 Quite High
medulla 35 53.28 Quite High
cva 7 42.88 Quite Low
Calcium channel 1 21.04 Low Low
anesthesia 2 5.00 Very Low Very Low Very Low
midbrain 1 5.00 Very Low Very Low Very Low
Inflammatory response 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Malaria 66 98.16 Very High Very High Very High
Infection 2 97.84 Very High Very High Very High
INFLAMMATION 2 93.60 High High
Shock 5 54.96 Quite High
Apoptosis 3 50.20 Quite High
Vasculitis 1 46.96 Quite Low
Cv General 3 Under Development 7 42.88 Quite Low
Thrombosis 1 23.20 Low Low
Injury 13 21.84 Low Low
Brain Injury 5 14.20 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
It can be envisaged that Epo induced eNOS could be protective in the context of severe malaria infection by preserving cerebral blood flow, facilitating effective collateral microcirculation, and anti-inflammatory effects.
Epo Positive_regulation (induced) of eNOS in blood associated with malaria, inflammation and infection
1) Confidence 0.08 Published 2009 Journal Malar J Section Body Doc Link PMC2785829 Disease Relevance 0.69 Pain Relevance 0.11

General Comments

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