INT289659

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Context Info
Confidence 0.02
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.82
Pain Relevance 1.13

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell proliferation (Tnfrsf1b) cell death (Tnfrsf1b) nucleus (Tnfrsf1b)
Tnfrsf1b (Mus musculus)
Mcpt3 (Mus musculus)
Pain Link Frequency Relevance Heat
cINOD 30 99.90 Very High Very High Very High
Inflammation 31 97.56 Very High Very High Very High
chemokine 4 95.96 Very High Very High Very High
Inflammatory mediators 3 81.04 Quite High
Inflammatory response 2 34.52 Quite Low
cytokine 11 5.28 Low Low
ketamine 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
INFLAMMATION 37 97.56 Very High Very High Very High
Pressure And Volume Under Development 10 92.64 High High
Lymphedema 45 83.92 Quite High
Inflammatory Bowel Disease 1 72.80 Quite High
Recurrence 1 71.60 Quite High
Increased Venous Pressure Under Development 3 50.00 Quite Low
Disease 5 5.00 Very Low Very Low Very Low
Cancer 3 5.00 Very Low Very Low Very Low
Necrosis 2 5.00 Very Low Very Low Very Low
Injury 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Both NSAID and sTNFR-1 treatment inhibited MCP-3 protein levels, supporting their function as generally anti-inflammatory.
sTNFR-1 Negative_regulation (inhibited) of MCP-3 associated with inflammation and cinod
1) Confidence 0.02 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2791214 Disease Relevance 0.82 Pain Relevance 1.13

General Comments

This test has worked.

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