INT289828

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Context Info
Confidence 0.07
First Reported 2005
Last Reported 2005
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.70
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (Bsnd) protein complex (Bsnd)
Bsnd (Mus musculus)
Pain Link Frequency Relevance Heat
fibrosis 7 44.16 Quite Low
gABA 4 5.00 Very Low Very Low Very Low
Glutamate 2 5.00 Very Low Very Low Very Low
fluoxetine 1 5.00 Very Low Very Low Very Low
adenocard 1 5.00 Very Low Very Low Very Low
Hippocampus 1 5.00 Very Low Very Low Very Low
antidepressant 1 5.00 Very Low Very Low Very Low
medulla 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Bartter Syndrome 3 99.40 Very High Very High Very High
Diabetes Mellitus 1 97.84 Very High Very High Very High
Acidosis 1 97.24 Very High Very High Very High
Hypomagnesemia 1 96.52 Very High Very High Very High
Hypocalcemia 2 95.76 Very High Very High Very High
Nonsyndromic Deafness 1 61.76 Quite High
Diabetes Insipidus 1 58.80 Quite High
Cystic Fibrosis 7 44.72 Quite Low
Disease 1 34.96 Quite Low
Targeted Disruption 6 14.36 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Functional loss of NKCC, Kir1.1, ClC-Kb or Barttin is a cause of Bartter syndrome, in which nephrogenic diabetes insipides, hypokalemia, hypomagnesemia and hypocalcemia have been observed as variations in phenotype [82–84].
Negative_regulation (loss) of Barttin associated with hypomagnesemia, hypocalcemia, diabetes mellitus and bartter syndrome
1) Confidence 0.07 Published 2005 Journal Cell Mol Life Sci Section Body Doc Link PMC2792346 Disease Relevance 0.70 Pain Relevance 0

General Comments

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