INT290389

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Context Info
Confidence 0.25
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.14
Pain Relevance 0.40

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell proliferation (CSF1R) signal transduction (CSF1R) plasma membrane (CSF1R)
CSF1R (Homo sapiens)
Pain Link Frequency Relevance Heat
Fibrositis 97 100.00 Very High Very High Very High
chemokine 12 86.76 High High
cytokine 12 86.36 High High
Inflammation 16 41.48 Quite Low
rheumatoid arthritis 5 33.60 Quite Low
Pain 9 33.08 Quite Low
antagonist 1 23.12 Low Low
Catechol-O-methyltransferase 4 5.00 Very Low Very Low Very Low
Catecholamine 2 5.00 Very Low Very Low Very Low
spastic colon 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Fibromyalgia 97 100.00 Very High Very High Very High
Infection 2 99.50 Very High Very High Very High
Disease 12 96.88 Very High Very High Very High
Disease Susceptibility 1 89.68 High High
Asthma 1 69.84 Quite High
INFLAMMATION 17 41.48 Quite Low
Fatigue 2 37.36 Quite Low
Autoimmune Disease 1 35.20 Quite Low
Rheumatoid Arthritis 5 33.60 Quite Low
Pain 10 33.08 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Given the large number of NLR genes and their growing number of disease associations, it is likely that more NLR gene variants will be found as risk factors for FMS, and that specific chronic infections will be identified that trigger FMS via their interactions with these genes.
Positive_regulation (trigger) of FMS Binding (interactions) of associated with disease, fibrositis and infection
1) Confidence 0.25 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2794536 Disease Relevance 1.14 Pain Relevance 0.40

General Comments

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