INT290430

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Context Info
Confidence 0.03
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.42
Pain Relevance 0.05

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (Mtor) mitochondrion (Mtor) Golgi apparatus (Mtor)
endoplasmic reticulum (Mtor) nucleus (Mtor) kinase activity (Mtor)
Anatomy Link Frequency
ventricle 2
Mtor (Mus musculus)
Pain Link Frequency Relevance Heat
fibrosis 10 99.10 Very High Very High Very High
anesthesia 3 45.28 Quite Low
ketamine 2 42.96 Quite Low
imagery 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Fibrosis 10 99.10 Very High Very High Very High
Stress 8 93.12 High High
Coronary Heart Disease 13 92.80 High High
Heart Disease 2 85.52 High High
Left Ventricular Hypertrophy 10 50.00 Quite Low
Hypertrophy 9 5.00 Very Low Very Low Very Low
Myocardial Infarction 3 5.00 Very Low Very Low Very Low
Targeted Disruption 2 5.00 Very Low Very Low Very Low
Congenital Anomalies 2 5.00 Very Low Very Low Very Low
Natriuresis 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The data indicate that depletion of MEF2C is sufficient to markedly attenuate the hypertrophic growth and myocardial fibrosis of overloaded left ventricle by a mechanism dependent on defective activation of mTOR/S6K pathway.
Regulation (dependent) of Positive_regulation (activation) of mTOR in ventricle associated with fibrosis
1) Confidence 0.03 Published 2009 Journal PLoS ONE Section Body Doc Link PMC2794538 Disease Relevance 0.42 Pain Relevance 0.05

General Comments

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