INT291224

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Context Info
Confidence 0.42
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 1.07
Pain Relevance 0.33

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (Map3k8) cell cycle (Map3k8) kinase activity (Map3k8)
cytoplasm (Map3k8)
Map3k8 (Mus musculus)
Pain Link Frequency Relevance Heat
cytokine 78 91.52 High High
Inflammatory mediators 4 79.12 Quite High
Inflammatory stimuli 10 75.60 Quite High
Inflammation 52 60.24 Quite High
tolerance 2 5.00 Very Low Very Low Very Low
anesthesia 2 5.00 Very Low Very Low Very Low
Kinase C 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Obesity 212 91.36 High High
INFLAMMATION 66 78.72 Quite High
Cancer 10 50.00 Quite Low
Necrosis 6 50.00 Quite Low
Diabetes Mellitus 16 49.92 Quite Low
Insulin Resistance 20 5.00 Very Low Very Low Very Low
Hypertension 2 5.00 Very Low Very Low Very Low
Targeted Disruption 2 5.00 Very Low Very Low Very Low
Impaired Glucose Tolerance 2 5.00 Very Low Very Low Very Low
Dislocations 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Furthermore, we showed that Tpl2 degradation was prevented following IKK?
Negative_regulation (prevented) of Protein_catabolism (degradation) of Tpl2
1) Confidence 0.42 Published 2010 Journal Diabetes Section Body Doc Link PMC2797946 Disease Relevance 0.65 Pain Relevance 0.16
or proteasome abolished Tpl2 degradation (Fig. 2C).


Negative_regulation (abolished) of Protein_catabolism (degradation) of Tpl2
2) Confidence 0.42 Published 2010 Journal Diabetes Section Body Doc Link PMC2797946 Disease Relevance 0.42 Pain Relevance 0.17

General Comments

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