INT291387

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Context Info
Confidence 0.12
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.34
Pain Relevance 0.45

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell differentiation (Cntf) cytosol (Cntf) extracellular space (Cntf)
growth (Cntf) plasma membrane (Slc1a2) nucleus (Cntf)
Cntf (Rattus norvegicus)
Slc1a2 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Glutamate 40 97.72 Very High Very High Very High
Glutamate receptor 6 58.64 Quite High
nMDA receptor 6 56.44 Quite High
cytokine 4 46.24 Quite Low
ketamine 2 5.00 Very Low Very Low Very Low
antagonist 2 5.00 Very Low Very Low Very Low
agonist 1 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Neurodegenerative Disease 3 76.16 Quite High
Disease 12 72.16 Quite High
Sclerosis 1 70.08 Quite High
Targeted Disruption 1 49.20 Quite Low
Stress 1 43.04 Quite Low
Leukemia 1 37.00 Quite Low
Drug Induced Neurotoxicity 1 11.48 Low Low
Neurological Disease 1 5.00 Very Low Very Low Very Low
Death 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Indeed, CNTF increases the glycosylation level of GLAST and GLT-1, and redistributes them into raft microdomains in which glutamate uptake is more efficient [31] leading to a net increase in the glutamate buffering capacity, while no changes were observed for EAAC1 [12], [31].
CNTF Positive_regulation (increases) of GLT-1 associated with glutamate
1) Confidence 0.12 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2798716 Disease Relevance 0.34 Pain Relevance 0.45

General Comments

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