INT291548

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Context Info
Confidence 0.02
First Reported 2010
Last Reported 2010
Negated 1
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.94
Pain Relevance 0.67

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Golgi apparatus (Gper, Sec22b) endoplasmic reticulum (Gper, Sec22b) vesicle-mediated transport (Sec22b)
plasma membrane (Gper) signal transducer activity (Gper)
Gper (Rattus norvegicus)
Sec22b (Rattus norvegicus)
Pain Link Frequency Relevance Heat
agonist 13 100.00 Very High Very High Very High
ischemia 54 98.76 Very High Very High Very High
Pyramidal cell 22 93.76 High High
Hippocampus 9 83.28 Quite High
withdrawal 8 72.20 Quite High
Neuronal excitability 5 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
nMDA receptor 1 5.00 Very Low Very Low Very Low
Glutamate 1 5.00 Very Low Very Low Very Low
antagonist 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Cv Unclassified Under Development 52 98.76 Very High Very High Very High
Cognitive Disorder 1 93.40 High High
Death 6 80.56 Quite High
Targeted Disruption 1 9.04 Low Low
Aging 4 5.00 Very Low Very Low Very Low
Reprotox - General 1 4 5.00 Very Low Very Low Very Low
Middle Cerebral Artery Infarction 4 5.00 Very Low Very Low Very Low
Stroke 4 5.00 Very Low Very Low Very Low
Injury 3 5.00 Very Low Very Low Very Low
Thrombosis 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Using the same experimental conditions (young female rats OVX 1 week before ischemia), we now show that a much lower dose of E2 and the selective GPR30 agonist G1, which does not bind to classical ERs [17], confer significant neuroprotection (Figure 2).
GPR30 Neg (not) Binding (bind) of ERs associated with ischemia and agonist
1) Confidence 0.02 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2799530 Disease Relevance 0.94 Pain Relevance 0.67

General Comments

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