INT291967

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Context Info
Confidence 0.00
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.98
Pain Relevance 0.14

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell differentiation (INA)
Anatomy Link Frequency
heart 2
INA (Homo sapiens)
NA (Homo sapiens)
Pain Link Frequency Relevance Heat
Action potential 16 74.36 Quite High
action potential duration 2 36.40 Quite Low
anesthesia 12 5.00 Very Low Very Low Very Low
adenocard 6 5.00 Very Low Very Low Very Low
agonist 4 5.00 Very Low Very Low Very Low
cocaine 2 5.00 Very Low Very Low Very Low
sodium channel 2 5.00 Very Low Very Low Very Low
beta blocker 2 5.00 Very Low Very Low Very Low
antagonist 2 5.00 Very Low Very Low Very Low
Catecholamine 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Congenital Anomalies 4 96.02 Very High Very High Very High
Heart Rate Under Development 2 95.72 Very High Very High Very High
Myocardial Infarction 8 93.52 High High
Heart Disease 2 84.28 Quite High
Shock 142 62.56 Quite High
Arrhythmias 2 Under Development 52 55.72 Quite High
Cv Unclassified Under Development 6 5.00 Very Low Very Low Very Low
Rheumatoid Arthritis 6 5.00 Very Low Very Low Very Low
Acidosis 4 5.00 Very Low Very Low Very Low
Pneumothorax 4 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In congestive heart failure, abnormalities in ion channels involved in automaticity ("funny" current If) , early after depolarizations (EADs) (IK1, IKs and the transient outward K current - Ito) and delayed afterdepolarizations (DADs) (late INa, INa L, increased Na+/Ca2+ exchanger function) promote arrhythmogenesis and may increase the DFT.
INa Positive_regulation (increased) of Na in heart associated with heart rate under development and congenital anomalies
1) Confidence 0.00 Published 2010 Journal Indian Pacing and Electrophysiology Journal Section Body Doc Link PMC2803603 Disease Relevance 0.49 Pain Relevance 0.07
In congestive heart failure, abnormalities in ion channels involved in automaticity ("funny" current If) , early after depolarizations (EADs) (IK1, IKs and the transient outward K current - Ito) and delayed afterdepolarizations (DADs) (late INa, INa L, increased Na+/Ca2+ exchanger function) promote arrhythmogenesis and may increase the DFT.
INa Positive_regulation (increased) of Na in heart associated with heart rate under development and congenital anomalies
2) Confidence 0.00 Published 2010 Journal Indian Pacing and Electrophysiology Journal Section Body Doc Link PMC2803603 Disease Relevance 0.49 Pain Relevance 0.07

General Comments

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