INT292448

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.15
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.28
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleus (PTK2, AKT1) plasma membrane (PTK2, AKT1) cytoplasm (PTK2, AKT1)
cytosol (PTK2, AKT1) cell differentiation (AKT1) nucleoplasm (AKT1)
Anatomy Link Frequency
AsPC-1 3
PTK2 (Homo sapiens)
AKT1 (Homo sapiens)
Pain Link Frequency Relevance Heat
palliative 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 37 96.32 Very High Very High Very High
Adhesions 7 69.28 Quite High
Pancreatic Cancer 48 50.00 Quite Low
Cancer 17 5.00 Very Low Very Low Very Low
Metastasis 4 5.00 Very Low Very Low Very Low
Malignant Neoplastic Disease 2 5.00 Very Low Very Low Very Low
Death 1 5.00 Very Low Very Low Very Low
Breast Cancer 1 5.00 Very Low Very Low Very Low
Lung Cancer 1 5.00 Very Low Very Low Very Low
Disease 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
These results indicated that in AsPC-1 cells, LN induced FAK and Akt phosphorylation in a time-dependent manner, and LN-induced Akt phosphorylation was mediated by FAK activation.


Positive_regulation (activation) of FAK in AsPC-1 Positive_regulation (mediated) of Phosphorylation (phosphorylation) of Akt in AsPC-1
1) Confidence 0.15 Published 2009 Journal Mol Cancer Section Body Doc Link PMC2806309 Disease Relevance 0.28 Pain Relevance 0

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox