INT292557

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Context Info
Confidence 0.08
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.52
Pain Relevance 0.22

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (F2RL1, MYD88) cytosol (MYD88) signal transduction (MYD88)
Golgi apparatus (F2RL1) intracellular (MYD88) cytoplasm (MYD88)
Anatomy Link Frequency
endothelial cells 1
F2RL1 (Homo sapiens)
MYD88 (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammation 10 97.92 Very High Very High Very High
Inflammatory response 1 94.80 High High
Inflammatory mediators 1 91.12 High High
cytokine 4 71.12 Quite High
IPN 1 36.52 Quite Low
Arthritis 1 35.76 Quite Low
agonist 13 5.00 Very Low Very Low Very Low
antagonist 5 5.00 Very Low Very Low Very Low
chemokine 1 5.00 Very Low Very Low Very Low
Potency 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
INFLAMMATION 12 97.92 Very High Very High Very High
Stress 2 77.24 Quite High
Reperfusion Injury 1 48.56 Quite Low
Cv Unclassified Under Development 1 47.80 Quite Low
Contact Dermatitis 1 39.72 Quite Low
Colitis 1 37.00 Quite Low
Inflammatory Pain 1 36.52 Quite Low
Arthritis 1 35.76 Quite Low
Disease 2 34.36 Quite Low
Cancer 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
pre-treatment has been shown to up-regulate functional PAR2 in endothelial cells [24,25], whilst recent studies have shown PAR2 to interact with TLR-4 signalling via myd88 [26], suggesting the potential for PAR2 to enhance inflammation.
PAR2 Binding (interact) of myd88 in endothelial cells associated with inflammation
1) Confidence 0.08 Published 2010 Journal Cellular Signalling Section Body Doc Link PMC2806525 Disease Relevance 0.52 Pain Relevance 0.22

General Comments

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