INT292562

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Context Info
Confidence 0.11
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.09
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

protein complex (TNFRSF1A) cytoplasm (TRADD) signal transducer activity (TRADD)
cytosol (TRADD) signal transduction (TRADD) extracellular space (TNFRSF1A)
TNFRSF1A (Homo sapiens)
TRADD (Homo sapiens)
Pain Link Frequency Relevance Heat
cytokine 8 49.28 Quite Low
Inflammation 20 46.56 Quite Low
agonist 26 23.36 Low Low
antagonist 10 5.00 Very Low Very Low Very Low
Inflammatory response 2 5.00 Very Low Very Low Very Low
Inflammatory mediators 2 5.00 Very Low Very Low Very Low
IPN 2 5.00 Very Low Very Low Very Low
chemokine 2 5.00 Very Low Very Low Very Low
Arthritis 2 5.00 Very Low Very Low Very Low
Potency 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 2 87.12 High High
INFLAMMATION 24 46.56 Quite Low
Cancer 4 5.00 Very Low Very Low Very Low
Disease 4 5.00 Very Low Very Low Very Low
Stress 4 5.00 Very Low Very Low Very Low
Arthritis 2 5.00 Very Low Very Low Very Low
Necrosis 2 5.00 Very Low Very Low Very Low
Cv Unclassified Under Development 2 5.00 Very Low Very Low Very Low
Reperfusion Injury 2 5.00 Very Low Very Low Very Low
Inflammatory Pain 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Upon activation TNFR1 associates with a number of proteins including TRADD, RIP, TRAF2, MADD and FADD [46].
TNFR1 Binding (associates) of TRADD
1) Confidence 0.11 Published 2010 Journal Cellular Signalling Section Body Doc Link PMC2806525 Disease Relevance 0.09 Pain Relevance 0
driven TNFR1 and TRADD complex formation (Panel B).


TNFR1 Binding (formation) of TRADD
2) Confidence 0.11 Published 2010 Journal Cellular Signalling Section Body Doc Link PMC2806525 Disease Relevance 0 Pain Relevance 0

General Comments

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