INT292575

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Context Info
Confidence 0.39
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.20
Pain Relevance 0.04

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular space (TNFRSF1A) extracellular region (TNFRSF1A) plasma membrane (TNFRSF1A)
nucleus (TNFRSF1A) protein complex (TNFRSF1A)
TNFRSF1A (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammation 10 74.32 Quite High
agonist 13 5.00 Very Low Very Low Very Low
antagonist 5 5.00 Very Low Very Low Very Low
cytokine 4 5.00 Very Low Very Low Very Low
Inflammatory response 1 5.00 Very Low Very Low Very Low
Inflammatory mediators 1 5.00 Very Low Very Low Very Low
IPN 1 5.00 Very Low Very Low Very Low
chemokine 1 5.00 Very Low Very Low Very Low
Arthritis 1 5.00 Very Low Very Low Very Low
Potency 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
INFLAMMATION 12 74.32 Quite High
Cancer 2 64.16 Quite High
Apoptosis 1 57.48 Quite High
Disease 2 41.84 Quite Low
Stress 2 5.00 Very Low Very Low Very Low
Arthritis 1 5.00 Very Low Very Low Very Low
Necrosis 1 5.00 Very Low Very Low Very Low
Cv Unclassified Under Development 1 5.00 Very Low Very Low Very Low
Reperfusion Injury 1 5.00 Very Low Very Low Very Low
Inflammatory Pain 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
At this point the exact mechanism mediating disruption is unclear, no studies to date have identified any PKC phosphorylation sites within the TNFR1 complex, and thus it is possible that additional intermediate proteins may be involved.
Phosphorylation (phosphorylation) of TNFR1
1) Confidence 0.39 Published 2010 Journal Cellular Signalling Section Body Doc Link PMC2806525 Disease Relevance 0.20 Pain Relevance 0.04

General Comments

This test has worked.

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