INT292713

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Context Info
Confidence 0.44
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 1.04
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (ABL1) protein modification process (ABL1) mitochondrion (ABL1)
nucleolus (ABL1) nucleus (ABL1) DNA binding (ABL1)
ABL1 (Homo sapiens)
Pain Link Frequency Relevance Heat
cytokine 3 5.00 Very Low Very Low Very Low
palliative 1 5.00 Very Low Very Low Very Low
chemokine 1 5.00 Very Low Very Low Very Low
Nicotine 1 5.00 Very Low Very Low Very Low
Inflammation 1 5.00 Very Low Very Low Very Low
qutenza 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Epilepsy 1 96.56 Very High Very High Very High
Apoptosis 29 91.76 High High
Myeloid Leukemia 37 90.72 High High
Philadelphia Chromosome 3 85.56 High High
Aging 2 80.96 Quite High
Chronic Myeloid Leukemia 12 50.00 Quite Low
Cancer 14 41.92 Quite Low
Leukemia 3 37.12 Quite Low
Stress 12 27.76 Quite Low
Myeloproliferative Disorder 45 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In addition, Okabe et al., demonstrated that FK228 treatment induces a diverse range of histone and non-histone protein acetylation in BCR-ABL transfected cell lines, including acetylation of p53, Hsp90 and BCR-ABL [81].
Phosphorylation (acetylation) of BCR-ABL
1) Confidence 0.44 Published 2010 Journal Invest New Drugs Section Body Doc Link PMC3003795 Disease Relevance 0.73 Pain Relevance 0
K562 cells exposed to 40┬ÁM Resveratrol for various time intervals ranging from 30min up to 12h failed to demonstrate any significant alteration of Bcr-Abl phosphorylation status (Fig 5).


Phosphorylation (phosphorylation) of Abl
2) Confidence 0.10 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2806839 Disease Relevance 0.31 Pain Relevance 0

General Comments

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