INT293200

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Context Info
Confidence 0.24
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.40
Pain Relevance 1.39

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (Grin1, Grin2b) embryo development (Grin2b) enzyme binding (Grin1)
cytoplasm (Grin1)
Grin1 (Rattus norvegicus)
Grin2b (Rattus norvegicus)
Pain Link Frequency Relevance Heat
long-term potentiation 6 98.44 Very High Very High Very High
nMDA receptor antagonist 3 92.28 High High
Antihyperalgesic 2 91.76 High High
Neuropathic pain 3 90.28 High High
antiallodynic 2 90.08 High High
Dorsal horn 10 88.80 High High
ketamine 35 88.16 High High
Ultiva 55 85.12 High High
spinal dorsal horn 9 84.20 Quite High
Antinociceptive 1 72.92 Quite High
Disease Link Frequency Relevance Heat
Neuropathic Pain 9 90.28 High High
Hyperalgesia 46 69.96 Quite High
Nociception 6 62.56 Quite High
Pain 6 5.00 Very Low Very Low Very Low
Post Operative Pain 5 5.00 Very Low Very Low Very Low
Injury 3 5.00 Very Low Very Low Very Low
INFLAMMATION 2 5.00 Very Low Very Low Very Low
Hypersensitivity 1 5.00 Very Low Very Low Very Low
Cancer Pain 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Furthermore, there were studies observed that increased NR2B tyrosine phosphorylation after LTP could blocked by MK-801 (an NMDAR channel blocker), suggesting that tyrosine phosphorylation of NR2B is triggered by activation of the NMDAR itself [13,14].
Positive_regulation (activation) of NMDAR Positive_regulation (triggered) of Phosphorylation (phosphorylation) of NR2B associated with long-term potentiation
1) Confidence 0.24 Published 2009 Journal Mol Pain Section Body Doc Link PMC2809057 Disease Relevance 0.40 Pain Relevance 1.39

General Comments

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