INT293461

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Context Info
Confidence 0.48
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 2
Disease Relevance 0
Pain Relevance 0.03

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transmembrane transport (Kcnh1)
Anatomy Link Frequency
synapse 1
Kcnh1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Action potential 6 51.92 Quite High
repetitive firing 2 41.92 Quite Low
Neuronal excitability 6 36.08 Quite Low
hyperexcitability 2 36.08 Quite Low
imagery 4 5.00 Very Low Very Low Very Low
tetrodotoxin 2 5.00 Very Low Very Low Very Low
Pyramidal cell 2 5.00 Very Low Very Low Very Low
potassium channel 2 5.00 Very Low Very Low Very Low
Hippocampus 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Infection 4 5.00 Very Low Very Low Very Low
Neurological Disease 2 5.00 Very Low Very Low Very Low
Channelopathies 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In turn this could be translated in conformational changes of Eag1 shaping the interaction with other yet to be discovered presynaptic proteins and hence its mobility.
Eag1 Binding (interaction) of
1) Confidence 0.48 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2810327 Disease Relevance 0 Pain Relevance 0.03
Mathematical analysis of their trajectories revealed how the motion of Eag1 gets restricted when the channels diffuse into the synapse, suggesting molecular interactions between Eag1 and synaptic components.
Eag1 Binding (interactions) of in synapse
2) Confidence 0.36 Published 2010 Journal PLoS ONE Section Abstract Doc Link PMC2810327 Disease Relevance 0 Pain Relevance 0

General Comments

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