INT294636

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Context Info
Confidence 0.02
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.62
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

molecular_function (Bcl2a1b) cellular_component (Bcl2a1b) biological_process (Bcl2a1b)
Bcl2a1b (Mus musculus)
Pain Link Frequency Relevance Heat
ischemia 18 50.00 Quite Low
cytokine 4 5.00 Very Low Very Low Very Low
Inflammation 4 5.00 Very Low Very Low Very Low
isoflurane 4 5.00 Very Low Very Low Very Low
anesthesia 2 5.00 Very Low Very Low Very Low
Buprenorphine 2 5.00 Very Low Very Low Very Low
Inflammatory response 2 5.00 Very Low Very Low Very Low
halothane 2 5.00 Very Low Very Low Very Low
ketamine 1 5.00 Very Low Very Low Very Low
fibrosis 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 43 97.20 Very High Very High Very High
Injury 40 81.60 Quite High
Cv Unclassified Under Development 18 50.00 Quite Low
Death 7 5.00 Very Low Very Low Very Low
INFLAMMATION 6 5.00 Very Low Very Low Very Low
Necrosis 4 5.00 Very Low Very Low Very Low
Adhesions 3 5.00 Very Low Very Low Very Low
Sepsis 3 5.00 Very Low Very Low Very Low
Myocardial Infarction 3 5.00 Very Low Very Low Very Low
Exsanguination 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Synthetic hBCL2-BH4 and hBCL2A1-BH4 peptides were both effective in reducing I/R, whereas a peptide from the BH3 domain of Bak, a pro-apoptotic BCL2 family member, was not (Figure 3).


Regulation (effective) of Gene_expression (peptides) of hBCL2A1-BH4 associated with apoptosis
1) Confidence 0.02 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2816997 Disease Relevance 0.62 Pain Relevance 0

General Comments

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