INT295037

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Context Info
Confidence 0.03
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 4
Disease Relevance 8.78
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell adhesion (Klra8) plasma membrane (Klra8)
Anatomy Link Frequency
E1a 2
A375 2
Klra8 (Mus musculus)
Pain Link Frequency Relevance Heat
palliative 4 5.00 Very Low Very Low Very Low
Inflammation 4 5.00 Very Low Very Low Very Low
Potency 4 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Cytomegalovirus Infection 264 100.00 Very High Very High Very High
Infection 104 100.00 Very High Very High Very High
Apoptosis 128 99.64 Very High Very High Very High
Cancer 372 95.04 Very High Very High Very High
Melanoma 60 94.28 High High
Disease 20 14.32 Low Low
Metastasis 40 5.00 Very Low Very Low Very Low
Death 20 5.00 Very Low Very Low Very Low
Toxicity 12 5.00 Very Low Very Low Very Low
Malignant Neoplastic Disease 12 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
As expected, with longer infection times, growth of A375 or B16 cells infected with Ad-CMV-E1a, Ad-hTERT-E1a, Ad-CMV-E1a-Apoptin and Ad-hTERT-E1a-Apoptin were inhibited.
Ad-CMV-E1a-Apoptin Binding (infected) of in E1a associated with cytomegalovirus infection and infection
1) Confidence 0.03 Published 2010 Journal Mol Cancer Section Body Doc Link PMC2818692 Disease Relevance 2.09 Pain Relevance 0
As shown in Figure 3, infection of all recombinant adenoviruses resulted in apoptosis of A375 and B16 cells, whereas in HEM cells, only infection with Ad-CMV-E1a and Ad-CMV-E1a-Apoptin induced apoptosis.
Ad-CMV-E1a-Apoptin Binding (infection) of in A375 associated with cytomegalovirus infection, apoptosis and infection
2) Confidence 0.03 Published 2010 Journal Mol Cancer Section Body Doc Link PMC2818692 Disease Relevance 2.32 Pain Relevance 0
As shown in Figure 3, infection of all recombinant adenoviruses resulted in apoptosis of A375 and B16 cells, whereas in HEM cells, only infection with Ad-CMV-E1a and Ad-CMV-E1a-Apoptin induced apoptosis.
Ad-CMV-E1a Binding (infection) of in A375 associated with cytomegalovirus infection, apoptosis and infection
3) Confidence 0.03 Published 2010 Journal Mol Cancer Section Body Doc Link PMC2818692 Disease Relevance 2.30 Pain Relevance 0
As expected, with longer infection times, growth of A375 or B16 cells infected with Ad-CMV-E1a, Ad-hTERT-E1a, Ad-CMV-E1a-Apoptin and Ad-hTERT-E1a-Apoptin were inhibited.
Ad-CMV-E1a Binding (infected) of in E1a associated with cytomegalovirus infection and infection
4) Confidence 0.03 Published 2010 Journal Mol Cancer Section Body Doc Link PMC2818692 Disease Relevance 2.07 Pain Relevance 0

General Comments

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