INT295616

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Context Info
Confidence 0.02
First Reported 2010
Last Reported 2010
Negated 0
Speculated 1
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.60
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

small molecule metabolic process (ACACB, INS) Golgi apparatus (ACACB) carbohydrate metabolic process (INS)
cell-cell signaling (INS) ligase activity (ACACB) cytoplasm (ACACB)
Anatomy Link Frequency
fat 1
ACACB (Homo sapiens)
INS (Homo sapiens)
Pain Link Frequency Relevance Heat
anesthesia 1 5.00 Very Low Very Low Very Low
alcohol 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Hyperinsulinism 1 99.30 Very High Very High Very High
Renal Disease 61 89.92 High High
Diabetes Mellitus 65 79.12 Quite High
Proteinuria 18 77.80 Quite High
Disease Susceptibility 6 59.40 Quite High
Congenital Anomalies 1 54.80 Quite High
Obesity 2 24.56 Low Low
Chronic Renal Failure 13 13.56 Low Low
Diabetic Retinopathy 5 5.00 Very Low Very Low Very Low
Disease 5 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Mice lacking Acacb have a normal life span, a higher rate of fatty acid oxidation, lower amounts of fat, and increased insulin sensitivity [19]–[21]; therefore, ACACB might affect insulin sensitivity via modulation of fatty acid metabolism.
ACACB Spec (might) Regulation (affect) of insulin in fat associated with hyperinsulinism
1) Confidence 0.02 Published 2010 Journal PLoS Genetics Section Body Doc Link PMC2820513 Disease Relevance 0.60 Pain Relevance 0

General Comments

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