INT295725

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Context Info
Confidence 0.05
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 1.92
Pain Relevance 0.32

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (PTGER2, RAC2) nuclear envelope (RAC2) intracellular (RAC2)
GTPase activity (RAC2) cytoplasm (RAC2) signal transducer activity (PTGER2)
Anatomy Link Frequency
endothelial cells 1
PTGER2 (Homo sapiens)
RAC2 (Homo sapiens)
Pain Link Frequency Relevance Heat
metalloproteinase 102 91.72 High High
cytokine 76 89.28 High High
Inflammation 40 86.12 High High
COX-2 inhibitor 23 57.92 Quite High
chemokine 20 5.00 Very Low Very Low Very Low
cINOD 4 5.00 Very Low Very Low Very Low
Inflammatory response 2 5.00 Very Low Very Low Very Low
fibrosis 1 5.00 Very Low Very Low Very Low
Bioavailability 1 5.00 Very Low Very Low Very Low
Inflammatory stimuli 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Herpes Simplex Virus 238 100.00 Very High Very High Very High
Infection 104 100.00 Very High Very High Very High
Adhesions 42 100.00 Very High Very High Very High
INFLAMMATION 44 86.12 High High
Sarcoma 79 83.52 Quite High
Apoptosis 8 68.24 Quite High
Vascular Neoplasms 1 44.48 Quite Low
Disease 5 32.40 Quite Low
Cancer 20 25.00 Low Low
Acquired Immune Deficiency Syndrome Or Hiv Infection 6 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
PGE2, secreted early during KSHV infection, profoundly increased the adhesion of uninfected endothelial cells to fibronectin by activating the small G protein Rac1.
PGE2 Positive_regulation (activating) of small G protein in endothelial cells associated with herpes simplex virus, adhesions and infection
1) Confidence 0.05 Published 2010 Journal PLoS Pathogens Section Abstract Doc Link PMC2820536 Disease Relevance 1.92 Pain Relevance 0.32

General Comments

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