INT296095

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Context Info
Confidence 0.14
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 3
Disease Relevance 4.41
Pain Relevance 1.94

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Acsm3) molecular_function (Acsm3) cellular_component (Acsm3)
ligase activity (Acsm3) biological_process (Acsm3) lipid metabolic process (Acsm3)
Anatomy Link Frequency
artery 6
Acsm3 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
cva 207 100.00 Very High Very High Very High
Inflammatory mediators 15 99.88 Very High Very High Very High
Inflammation 36 89.04 High High
cytokine 27 71.96 Quite High
Inflammatory response 12 13.92 Low Low
ketamine 6 5.00 Very Low Very Low Very Low
anesthesia 3 5.00 Very Low Very Low Very Low
withdrawal 3 5.00 Very Low Very Low Very Low
adenocard 3 5.00 Very Low Very Low Very Low
Kinase C 3 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Subarachnoid Hemorrhage 189 100.00 Very High Very High Very High
Hemorrhage 18 100.00 Very High Very High Very High
Increased Venous Pressure Under Development 69 99.96 Very High Very High Very High
INFLAMMATION 51 99.68 Very High Very High Very High
Vasculitis 12 89.76 High High
Stress 54 86.88 High High
Thrombosis Related Under Development 6 18.88 Low Low
Adhesions 6 6.16 Low Low
Death 6 5.00 Very Low Very Low Very Low
Body Weight 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The main findings of this study are that (1) vascular inflammatory mediators were induced after SAH and could be remarkably repressed when treated with melatonin; (2) the increased lipid peroxidation in the artery tissues could be significantly downregulated after melatonin injections following experimental SAH; (3) after melatonin administration, the postSAH reduced antioxidative status was ameliorated in this two-hemorrhage model; (4) in agreement with the previous research [5], treatment with melatonin prevented cerebral vasospasm of the basilar arteries in rabbits.
Negative_regulation (repressed) of Positive_regulation (induced) of SAH in artery associated with inflammatory mediators, cva and increased venous pressure under development
1) Confidence 0.14 Published 2009 Journal Mediators of Inflammation Section Body Doc Link PMC2821769 Disease Relevance 1.13 Pain Relevance 0.52
The main findings of this study are that (1) vascular inflammatory mediators were induced after SAH and could be remarkably repressed when treated with melatonin; (2) the increased lipid peroxidation in the artery tissues could be significantly downregulated after melatonin injections following experimental SAH; (3) after melatonin administration, the postSAH reduced antioxidative status was ameliorated in this two-hemorrhage model; (4) in agreement with the previous research [5], treatment with melatonin prevented cerebral vasospasm of the basilar arteries in rabbits.
Negative_regulation (reduced) of Positive_regulation (induced) of SAH in artery associated with inflammatory mediators, cva and increased venous pressure under development
2) Confidence 0.14 Published 2009 Journal Mediators of Inflammation Section Body Doc Link PMC2821769 Disease Relevance 1.49 Pain Relevance 0.65
The main findings of this study are that (1) vascular inflammatory mediators were induced after SAH and could be remarkably repressed when treated with melatonin; (2) the increased lipid peroxidation in the artery tissues could be significantly downregulated after melatonin injections following experimental SAH; (3) after melatonin administration, the postSAH reduced antioxidative status was ameliorated in this two-hemorrhage model; (4) in agreement with the previous research [5], treatment with melatonin prevented cerebral vasospasm of the basilar arteries in rabbits.
Negative_regulation (prevented) of Positive_regulation (induced) of SAH in artery associated with inflammatory mediators, cva and increased venous pressure under development
3) Confidence 0.14 Published 2009 Journal Mediators of Inflammation Section Body Doc Link PMC2821769 Disease Relevance 1.80 Pain Relevance 0.77

General Comments

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