INT296929

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Context Info
Confidence 0.00
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.43
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Casq1) Golgi apparatus (Casq1) molecular_function (Casq1)
response to stress (Casq1) cytoplasm (Casq1)
Casq1 (Rattus norvegicus)
Tmie (Mus musculus)
Pain Link Frequency Relevance Heat
ischemia 2 5.00 Very Low Very Low Very Low
tolerance 1 5.00 Very Low Very Low Very Low
Glutamate 1 5.00 Very Low Very Low Very Low
Inflammation 1 5.00 Very Low Very Low Very Low
Morphine 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Hypoxia 52 90.36 High High
Stress 6 63.68 Quite High
Apoptosis 25 61.60 Quite High
Shock 1 50.08 Quite High
Death 4 43.36 Quite Low
Myocardial Infarction 4 31.76 Quite Low
Injury 16 15.56 Low Low
Cv Unclassified Under Development 2 5.00 Very Low Very Low Very Low
B-cell Acute Lymphoblastic Leukemia 1 5.00 Very Low Very Low Very Low
Repression 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Considering that calsequestrin overexpression increases SR Ca2+ content by inhibition of RyRs,42 calsequestrin overexpression is also likely to contribute to intracellular Ca2+ overload.
Gene_expression (overexpression) of calsequestrin Positive_regulation (increases) of SR
1) Confidence 0.00 Published 2010 Journal Yonsei Medical Journal Section Body Doc Link PMC2824862 Disease Relevance 0.43 Pain Relevance 0

General Comments

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