INT296968

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Context Info
Confidence 0.61
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 3
Disease Relevance 2.48
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (CIDEA) cell death (CIDEA) lipid particle (CIDEA)
nucleus (CIDEA) intracellular (CIDEA) lipid metabolic process (CIDEA)
CIDEA (Homo sapiens)
Pain Link Frequency Relevance Heat
tolerance 3 5.00 Very Low Very Low Very Low
anesthesia 3 5.00 Very Low Very Low Very Low
behavioral therapy 3 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Obesity 375 94.40 High High
Hyperinsulinism 3 88.64 High High
Targeted Disruption 27 86.96 High High
Weight Loss 6 5.00 Very Low Very Low Very Low
Impaired Glucose Tolerance 3 5.00 Very Low Very Low Very Low
Body Weight 3 5.00 Very Low Very Low Very Low
Thinness 3 5.00 Very Low Very Low Very Low
Death 3 5.00 Very Low Very Low Very Low
Disease 3 5.00 Very Low Very Low Very Low
Gallstones 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Altogether, RIP140 had a smaller impact on CIDEA mRNA levels in humans as compared to mice.
Transcription (levels) of CIDEA
1) Confidence 0.61 Published 2010 Journal BMC Endocr Disord Section Body Doc Link PMC2825205 Disease Relevance 0.90 Pain Relevance 0
In a one sided analysis, which is justified for confirmatory data, CIDEA mRNA levels were induced significantly by RIP140 silencing (P < 0.05).
Transcription (levels) of CIDEA
2) Confidence 0.53 Published 2010 Journal BMC Endocr Disord Section Body Doc Link PMC2825205 Disease Relevance 0.87 Pain Relevance 0
A trend toward increased mRNA levels for GLUT1 and CIDEA was observed (P = 0.0594 and P = 0.052, respectively).
Transcription (levels) of CIDEA
3) Confidence 0.45 Published 2010 Journal BMC Endocr Disord Section Body Doc Link PMC2825205 Disease Relevance 0.70 Pain Relevance 0

General Comments

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