INT298103

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.03
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (Cav1, Ca2) cytosol (Cav1, Ca2) endosome (Cav1)
mitochondrion (Cav1) lyase activity (Ca2) Golgi apparatus (Cav1)
Cav1 (Rattus norvegicus)
Ca2 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
addiction 6 5.00 Very Low Very Low Very Low
Migraine 2 5.00 Very Low Very Low Very Low
agonist 1 5.00 Very Low Very Low Very Low
sodium channel 1 5.00 Very Low Very Low Very Low
antagonist 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Shock 4 41.28 Quite Low
Channelopathies 8 15.52 Low Low
Syndrome 4 5.00 Very Low Very Low Very Low
Disease 3 5.00 Very Low Very Low Very Low
Congenital Anomalies 2 5.00 Very Low Very Low Very Low
Migraine With Aura 1 5.00 Very Low Very Low Very Low
Headache 1 5.00 Very Low Very Low Very Low
Ataxia 1 5.00 Very Low Very Low Very Low
Autism 1 5.00 Very Low Very Low Very Low
Cytomegalovirus Infection 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Evidence for this mechanism comes from single-channel Ca2+ records of CaV1.2, in which CDI reduces the frequency of opening without completely silencing channels (Imredy and Yue, 1994).
CaV1 Binding (records) of Ca2
1) Confidence 0.03 Published 2010 Journal The Journal of General Physiology Section Body Doc Link PMC2828906 Disease Relevance 0 Pain Relevance 0

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox