INT298472

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Context Info
Confidence 0.05
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.41
Pain Relevance 0.32

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular region (LPA, CXCL16) extracellular space (CXCL16) peptidase activity (LPA)
transport (LPA) small molecule metabolic process (LPA) plasma membrane (CXCL16)
Anatomy Link Frequency
macrophages 2
LPA (Homo sapiens)
CXCL16 (Homo sapiens)
Pain Link Frequency Relevance Heat
Multiple sclerosis 82 97.80 Very High Very High Very High
Potency 2 90.12 High High
agonist 10 88.96 High High
aspirin 5 73.80 Quite High
Inflammation 38 34.52 Quite Low
cINOD 2 25.20 Quite Low
chemokine 35 5.00 Very Low Very Low Very Low
5HT 23 5.00 Very Low Very Low Very Low
metalloproteinase 15 5.00 Very Low Very Low Very Low
cytokine 9 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Multiple Sclerosis 93 97.80 Very High Very High Very High
Adhesions 4 96.40 Very High Very High Very High
Increased Venous Pressure Under Development 2 71.04 Quite High
Neurological Disease 2 48.24 Quite Low
Disease 30 34.88 Quite Low
INFLAMMATION 40 34.52 Quite Low
Cancer 4 29.92 Quite Low
Metastasis 1 29.36 Quite Low
Neurodegenerative Disease 8 5.00 Very Low Very Low Very Low
Thrombosis 6 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The study of Kang et al [145] documents augmented production of CXCL16, a regulator of T cell migration, by macrophages due to presence of LPA (or S1P) following stimulation by LPS, of interest in MS because of parallels with S1P.
LPA Positive_regulation (augmented) of Gene_expression (production) of CXCL16 in macrophages associated with multiple sclerosis
1) Confidence 0.05 Published 2010 Journal J Neuroinflammation Section Body Doc Link PMC2829540 Disease Relevance 0.41 Pain Relevance 0.32

General Comments

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