INT298758

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Context Info
Confidence 0.57
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 0.12
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (ATP5A1) small molecule metabolic process (ATP5A1) transmembrane transporter activity (ATP5A1)
ATPase activity (ATP5A1) plasma membrane (ATP5A1) embryo development (ATP5A1)
Anatomy Link Frequency
tectal lobes 1
ATP5A1 (Homo sapiens)
Pain Link Frequency Relevance Heat
Pain 6 5.00 Very Low Very Low Very Low
halothane 2 5.00 Very Low Very Low Very Low
peripheral neuropathy 2 5.00 Very Low Very Low Very Low
antiepileptic Drug 1 5.00 Very Low Very Low Very Low
backache 1 5.00 Very Low Very Low Very Low
lidocaine 1 5.00 Very Low Very Low Very Low
addiction 1 5.00 Very Low Very Low Very Low
agonist 1 5.00 Very Low Very Low Very Low
Glutamate 1 5.00 Very Low Very Low Very Low
positron emission tomography 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Syndrome 3 59.04 Quite High
Mitochondrial Disorders 1 58.68 Quite High
Parkinson's Disease 4 49.68 Quite Low
Muscle Disease 23 48.72 Quite Low
Disease 31 5.00 Very Low Very Low Very Low
Coronary Heart Disease 17 5.00 Very Low Very Low Very Low
Glycogen Storage Disease 10 5.00 Very Low Very Low Very Low
Hypoglycemia 10 5.00 Very Low Very Low Very Low
Muscle Weakness 10 5.00 Very Low Very Low Very Low
Myoglobinuria 8 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Using this technique, we have shown that secondary downregulation of ATPsynthase (complex V) occurs in response to reduced electron flow across complexes I–IV [145].
Negative_regulation (downregulation) of ATPsynthase
1) Confidence 0.57 Published 2010 Journal Journal of Biomedicine and Biotechnology Section Body Doc Link PMC2877206 Disease Relevance 0.12 Pain Relevance 0
But little is known about the neuronal substrate of the OMR and obtained results were discussed controversially: Springer et al. [14] showed that the OMR of goldfish depends upon an intact tectum opticum: after an ablation of both tectal lobes the OMR of goldfish was completely abolished.
Negative_regulation (abolished) of OMR in tectal lobes
2) Confidence 0.26 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2830482 Disease Relevance 0 Pain Relevance 0

General Comments

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