INT299295

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Context Info
Confidence 0.00
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.22
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell differentiation (AKT1) nucleoplasm (AKT1) transport (AKT1)
small molecule metabolic process (AKT1) enzyme binding (AKT1) carbohydrate metabolic process (AKT1)
OR3A4P (Homo sapiens)
AKT1 (Homo sapiens)
Pain Link Frequency Relevance Heat
Morphine 3 13.28 Low Low
analgesia 2 11.40 Low Low
opiate 9 10.84 Low Low
palliative 2 8.68 Low Low
antagonist 10 5.00 Very Low Very Low Very Low
mu opioid receptor 5 5.00 Very Low Very Low Very Low
Opioid 5 5.00 Very Low Very Low Very Low
Immobilon 2 5.00 Very Low Very Low Very Low
narcan 1 5.00 Very Low Very Low Very Low
Pain management 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Hyperinsulinism 1 92.16 High High
Renal Cancer 2 64.40 Quite High
Disease 2 62.48 Quite High
Cancer 6 5.00 Very Low Very Low Very Low
Pain 2 5.00 Very Low Very Low Very Low
Anaemia 1 5.00 Very Low Very Low Very Low
Thrombocytopenia 1 5.00 Very Low Very Low Very Low
Hepatocellular Cancer 1 5.00 Very Low Very Low Very Low
Acquired Immune Deficiency Syndrome Or Hiv Infection 1 5.00 Very Low Very Low Very Low
Asthenia 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
This study extends these finding by demonstrating that the potent protein tyrosine phosphatase inhibitor, 3,4-Dephostatin, blocks MNTX inhibition of VEGF-induced Src and Akt phosphorylation. 3,4-Dephostatin is known to block the phosphatase activity of PTP-1B, SHPTP-1 and CD45 [20,40,46-48].
3,4-Dephostatin Negative_regulation (block) of Phosphorylation (phosphorylation) of Akt
1) Confidence 0.00 Published 2010 Journal J Angiogenes Res Section Body Doc Link PMC2831839 Disease Relevance 0.22 Pain Relevance 0

General Comments

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