INT299552

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Context Info
Confidence 0.00
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.12
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

aging (TGFB1) enzyme binding (TGFB1) embryo development (TGFB1)
cytoplasm (TGFB1) extracellular space (TGFB1) extracellular region (TGFB1)
Anatomy Link Frequency
astrocyte 2
TGFB1 (Homo sapiens)
Alk (Rattus norvegicus)
Pain Link Frequency Relevance Heat
antagonist 5 46.40 Quite Low
Glutamate 4 28.96 Quite Low
ischemia 7 18.64 Low Low
cytokine 17 5.00 Very Low Very Low Very Low
ketamine 2 5.00 Very Low Very Low Very Low
anesthesia 2 5.00 Very Low Very Low Very Low
Multiple sclerosis 1 5.00 Very Low Very Low Very Low
isoflurane 1 5.00 Very Low Very Low Very Low
imagery 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Injury 35 65.44 Quite High
Glioblastoma 1 38.60 Quite Low
Death 6 32.92 Quite Low
Cv Unclassified Under Development 6 18.64 Low Low
Hypoxia 10 18.28 Low Low
Gliosis 6 5.00 Very Low Very Low Very Low
Infection 3 5.00 Very Low Very Low Very Low
Stress 2 5.00 Very Low Very Low Very Low
Multiple Sclerosis 2 5.00 Very Low Very Low Very Low
Cv General 4 Under Development 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
We hypothesized that TGFß1 would act though ALK1 to induce astrocyte specification; however, TGFß1 only increased Smad 2/3 phosphorylation, reflecting signaling though ALK5 instead of ALK1.
TGF Positive_regulation (increased) of Phosphorylation (phosphorylation) of ALK5 in astrocyte
1) Confidence 0.00 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2832687 Disease Relevance 0.12 Pain Relevance 0

General Comments

This test has worked.

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