INT299673

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Context Info
Confidence 0.01
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Anatomy Link Frequency
cleavage 1
ECB2 (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammation 8 5.00 Very Low Very Low Very Low
cINOD 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Cellulitis 1 37.68 Quite Low
INFLAMMATION 9 5.00 Very Low Very Low Very Low
Infection 8 5.00 Very Low Very Low Very Low
Disease 3 5.00 Very Low Very Low Very Low
Hemolysis 3 5.00 Very Low Very Low Very Low
Communicable Diseases 2 5.00 Very Low Very Low Very Low
Targeted Disruption 2 5.00 Very Low Very Low Very Low
Sprains And Strains 1 5.00 Very Low Very Low Very Low
Bacterial Infection 1 5.00 Very Low Very Low Very Low
Pneumonia 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Another mechanism to prevent recognition is by inactivating IgG, either directly by proteases IdeS, Mac-2, and SpeB, or indirectly by SAK-activated plasmin. b There are three ways by which S. aureus and GAS modulate opsonization: (1) C3 cleavage; directly by SpeB or indirectly by SAK-mediated activated plasmin ClfA binds human fI, thereby enhancing C3 cleavage. (2) Convertase modulation; S. aureus inactivates the C3 convertases by secreting SCIN, SCIN-B, SCIN-C, Efb, and Ecb. (3) Modulating host regulators; Sbi and the streptococcal M-protein bind the negative convertase regulators (human FH, FHL1, C4BP, and CD46) while SpeB cleaves the positive convertase regulator, properdin
Localization (secreting) of Ecb in cleavage
1) Confidence 0.01 Published 2010 Journal J Mol Med Section Body Doc Link PMC2832872 Disease Relevance 0 Pain Relevance 0

General Comments

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