INT299983

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Context Info
Confidence 0.09
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 4
Disease Relevance 3.24
Pain Relevance 0.28

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

methyltransferase activity (FTSJ3) nucleolus (FTSJ3) nucleus (FTSJ3)
Anatomy Link Frequency
bone marrow 2
FTSJ3 (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammation 24 91.80 High High
Bioavailability 8 68.12 Quite High
Angina 8 65.16 Quite High
ischemia 16 60.12 Quite High
Nicotine 16 47.76 Quite Low
cytokine 4 15.44 Low Low
agonist 24 5.00 Very Low Very Low Very Low
depression 4 5.00 Very Low Very Low Very Low
antagonist 4 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Aging 12 99.56 Very High Very High Very High
Left Ventricular Dysfunction 8 98.98 Very High Very High Very High
Pressure And Volume Under Development 8 96.72 Very High Very High Very High
Hyperlipidemia 8 96.64 Very High Very High Very High
Heart Rate Under Development 12 94.64 High High
Myocardial Infarction 92 92.68 High High
INFLAMMATION 24 91.80 High High
Apoptosis 8 87.32 High High
Disease 12 85.28 High High
Coronary Artery Disease 108 82.16 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
These data are in accordance with observations in animal model that statin-induced prevention of left ventricular dysfunction was strongly associated the ability of statins to mobilize EPCs [54].
Localization (mobilize) of EPCs associated with left ventricular dysfunction
1) Confidence 0.09 Published 2010 Journal J Angiogenes Res Section Body Doc Link PMC2834645 Disease Relevance 1.42 Pain Relevance 0.17
Angiotensin II accelerates the onset of EPC senescence, leading to impaired proliferation of EPCs; this seems to be inhibited by treatment with the angiotensin II type 1 receptor blocker, valsartan [29].
Localization (proliferation) of EPCs associated with aging
2) Confidence 0.08 Published 2010 Journal J Angiogenes Res Section Body Doc Link PMC2834645 Disease Relevance 0.58 Pain Relevance 0.03
Indeed, the mobilization of EPCs from the bone marrow may be promptly stimulated by cardiopulmonary bypass but the extent of such mobilisation is significantly affected by pre-existing risk factors and the European System for Cardiac Operative Risk Evaluation (EuroSCORE) score.
Localization (mobilization) of EPCs in bone marrow
3) Confidence 0.08 Published 2010 Journal J Angiogenes Res Section Body Doc Link PMC2834645 Disease Relevance 0.44 Pain Relevance 0.04
Hypercholesterolaemia may also directly affect the bone marrow, resulting in depletion or exhaustion of the bone marrow pool of endothelial progenitors, with a consequent limited supply of EPCs released into circulation [23].
Localization (released) of EPCs in bone marrow associated with hyperlipidemia
4) Confidence 0.08 Published 2010 Journal J Angiogenes Res Section Body Doc Link PMC2834645 Disease Relevance 0.71 Pain Relevance 0.03

General Comments

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