INT301281

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.22
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.34
Pain Relevance 0.21

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (Tnfsf11, Clec5a) cell differentiation (Tnfsf11) extracellular space (Tnfsf11)
extracellular region (Tnfsf11) intracellular (Tnfsf11) cytoplasm (Tnfsf11)
Anatomy Link Frequency
osteoclast 2
Tnfsf11 (Mus musculus)
Clec5a (Mus musculus)
Pain Link Frequency Relevance Heat
agonist 20 86.64 High High
cytokine 7 61.96 Quite High
Arthritis 37 51.96 Quite High
Inflammation 36 50.00 Quite Low
rheumatoid arthritis 8 5.00 Very Low Very Low Very Low
antagonist 4 5.00 Very Low Very Low Very Low
imagery 3 5.00 Very Low Very Low Very Low
Pain 2 5.00 Very Low Very Low Very Low
Inflammatory response 2 5.00 Very Low Very Low Very Low
fibrosis 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Hypercalcemia 7 98.96 Very High Very High Very High
Arthritis 53 83.24 Quite High
Pressure And Volume Under Development 9 53.84 Quite High
INFLAMMATION 30 50.00 Quite Low
Disease 22 26.28 Quite Low
Rheumatoid Arthritis 8 5.00 Very Low Very Low Very Low
Arthropathy 5 5.00 Very Low Very Low Very Low
Dengue 3 5.00 Very Low Very Low Very Low
Osteoporosis 2 5.00 Very Low Very Low Very Low
Injury 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The apparent regulation of the TRAP, RANKL, ATPV0D2, and MMP9 expression after antiĀ–MDL-1 treatment suggests MDL-1 receptor activation may promote osteoclast development and function.
RANKL Regulation (regulation) of Gene_expression (expression) of MDL-1 in osteoclast
1) Confidence 0.22 Published 2010 Journal The Journal of Experimental Medicine Section Body Doc Link PMC2839155 Disease Relevance 0.34 Pain Relevance 0.21

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox