INT302434

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Context Info
Confidence 0.05
First Reported 2010
Last Reported 2010
Negated 1
Speculated 1
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 1.11
Pain Relevance 1.22

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

endosome (Prf1) extracellular region (Prf1) cytoplasmic membrane-bounded vesicle (Prf1)
plasma membrane (Prf1) transmembrane transport (Prf1)
Anatomy Link Frequency
pore 2
Prf1 (Mus musculus)
Pain Link Frequency Relevance Heat
Pain 92 100.00 Very High Very High Very High
Lasting pain 23 100.00 Very High Very High Very High
nMDA receptor 10 77.24 Quite High
long-term potentiation 11 70.32 Quite High
Calcium channel 8 61.68 Quite High
corticosteroid 98 53.12 Quite High
dexamethasone 3 43.24 Quite Low
Pain threshold 9 36.76 Quite Low
agonist 3 15.56 Low Low
Opioid 6 14.00 Low Low
Disease Link Frequency Relevance Heat
Pain 120 100.00 Very High Very High Very High
Sprains And Strains 2 73.84 Quite High
Nociception 7 32.56 Quite Low
Stress 45 5.00 Very Low Very Low Very Low
Depression 16 5.00 Very Low Very Low Very Low
Cognitive Disorder 10 5.00 Very Low Very Low Very Low
Shock 7 5.00 Very Low Very Low Very Low
Anxiety Disorder 7 5.00 Very Low Very Low Very Low
Low Back Pain 5 5.00 Very Low Very Low Very Low
Neurodegenerative Disease 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Analyses investigating the moderating effect of chronic pain complaints on the effect of the BDNF Met allele, revealed a significant interaction between pain status and the BDNF Met allele in the model of the P1- component of Fz (B?
Spec (investigating) Regulation (effect) of Transcription (model) of P1 associated with pain and lasting pain
1) Confidence 0.05 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2964315 Disease Relevance 1.03 Pain Relevance 1.03
On the other hand, Chameau et al. [105] found by quantitative PCR that corticosterone did not change the mRNA expression of the pore-forming Cav1 subunit of the L-type Ca2+ channel, and ruled out transcriptional mechanisms in the effects they observed.
Neg (not) Regulation (change) of Transcription (expression) of pore-forming Cav1 subunit in pore
2) Confidence 0.00 Published 2010 Journal Mol Brain Section Body Doc Link PMC2841592 Disease Relevance 0.07 Pain Relevance 0.19

General Comments

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