INT302632

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Context Info
Confidence 0.37
First Reported 2010
Last Reported 2010
Negated 0
Speculated 1
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.21
Pain Relevance 0.05

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

intracellular (Rac1, Rhog) GTPase activity (Rac1, Rhog) plasma membrane (Rac1, Rhog)
enzyme binding (Rac1) cytoplasm (Rac1) cell proliferation (Rac1)
Anatomy Link Frequency
endothelial cells 2
Rac1 (Mus musculus)
Rhog (Mus musculus)
Pain Link Frequency Relevance Heat
cINOD 1 50.68 Quite High
ischemia 2 5.00 Very Low Very Low Very Low
antagonist 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Cancer 85 83.60 Quite High
INFLAMMATION 1 50.44 Quite High
Targeted Disruption 6 5.00 Very Low Very Low Very Low
Colorectal Cancer 4 5.00 Very Low Very Low Very Low
Cv Unclassified Under Development 4 5.00 Very Low Very Low Very Low
Metastasis 3 5.00 Very Low Very Low Very Low
Pathologic Neovascularization 3 5.00 Very Low Very Low Very Low
Adhesions 2 5.00 Very Low Very Low Very Low
Injury 2 5.00 Very Low Very Low Very Low
Hemangiosarcoma 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Although it is tempting to speculate that RhoG activity may compensate for Rac1 in wild-type endothelial cells, thus explaining the reduced requirement for Rac1 in wild-type angiogenic events in vivo, this is also not likely because Rac1-deletion in both ?
Rac1 Spec (may) Positive_regulation (compensate) of Gene_expression (activity) of RhoG in endothelial cells
1) Confidence 0.37 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2842301 Disease Relevance 0.21 Pain Relevance 0.05

General Comments

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