INT302675

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Context Info
Confidence 0.15
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.73
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (PLAA) mitochondrion (OPA1) mitochondrion organization (OPA1)
GTPase activity (OPA1)
OPA1 (Homo sapiens)
PLAA (Homo sapiens)
Pain Link Frequency Relevance Heat
Multiple sclerosis 20 5.00 Very Low Very Low Very Low
peripheral neuropathy 16 5.00 Very Low Very Low Very Low
Migraine 8 5.00 Very Low Very Low Very Low
headache 6 5.00 Very Low Very Low Very Low
Inflammation 4 5.00 Very Low Very Low Very Low
Spinal cord 2 5.00 Very Low Very Low Very Low
adenocard 2 5.00 Very Low Very Low Very Low
Electroencephalography 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Disease 40 77.20 Quite High
Ganglion Cysts 10 74.08 Quite High
Nerve Degeneration 2 71.76 Quite High
Parkinson's Disease 6 65.28 Quite High
Neurological Disease 4 50.00 Quite Low
Apoptosis 6 48.96 Quite Low
Congenital Anomalies 8 5.88 Low Low
Optic Atrophy 30 5.00 Very Low Very Low Very Low
Demyelinating Disease 24 5.00 Very Low Very Low Very Low
Chronic Progressive External Ophthalmoplegia 22 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Although these remain to be proven experimentally, a gain of function mechanism cannot explain the association of OPA1 deletions and splice-site mutations with DOA+, both of which lead to haploinsufficiency, and were identified in DOA+ families in this study.
OPA1 Binding (association) of DOA
1) Confidence 0.15 Published 2010 Journal Brain Section Body Doc Link PMC2842512 Disease Relevance 0.36 Pain Relevance 0
Although these remain to be proven experimentally, a gain of function mechanism cannot explain the association of OPA1 deletions and splice-site mutations with DOA+, both of which lead to haploinsufficiency, and were identified in DOA+ families in this study.
OPA1 Binding (association) of DOA
2) Confidence 0.15 Published 2010 Journal Brain Section Body Doc Link PMC2842512 Disease Relevance 0.36 Pain Relevance 0

General Comments

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