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Context Info
Confidence 0.37
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.67
Pain Relevance 0.08

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (MPV17) homeostatic process (MPV17) peroxisome (MPV17)
molecular_function (MPV17)
MPV17 (Homo sapiens)
Pain Link Frequency Relevance Heat
adenocard 1 76.72 Quite High
peripheral neuropathy 8 73.60 Quite High
Multiple sclerosis 10 5.00 Very Low Very Low Very Low
Migraine 4 5.00 Very Low Very Low Very Low
headache 3 5.00 Very Low Very Low Very Low
Inflammation 2 5.00 Very Low Very Low Very Low
Spinal cord 1 5.00 Very Low Very Low Very Low
Electroencephalography 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Disease 20 86.56 High High
Syndrome 6 82.48 Quite High
Liver Failure 1 77.76 Quite High
Epilepsy 1 76.16 Quite High
Ataxia 11 74.24 Quite High
Peripheral Neuropathy 8 73.60 Quite High
Muscle Disease 9 71.52 Quite High
Chronic Progressive External Ophthalmoplegia 11 69.76 Quite High
Mitochondrial Disorders 4 56.48 Quite High
Neurological Disease 2 50.00 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Over the past decade, a growing number of nuclear genetic defects have been identified that disrupt mitochondrial function either by a reduction in mtDNA copy number, and/or the accumulation of secondary mtDNA deletions: POLG1, POLG2, PEO1, SLC25A4, TYMP, DGUOK, TK2, SUCLA2, SUCLG1, MPV17 and RRM2B (Hudson and Chinnery, 2006; Chinnery and Zeviani, 2008; Copeland, 2008).
Negative_regulation (reduction) of Localization (accumulation) of MPV17
1) Confidence 0.37 Published 2010 Journal Brain Section Body Doc Link PMC2842512 Disease Relevance 0.67 Pain Relevance 0.08

General Comments

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