INT302807

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Context Info
Confidence 0.04
First Reported 2009
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.93
Pain Relevance 0.23

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (Ripk1) mitochondrion (Ripk1) plasma membrane (Ripk1)
protein complex (Ripk1) kinase activity (Ripk1) cytoplasm (Ripk1)
Ripk1 (Mus musculus)
Pain Link Frequency Relevance Heat
rheumatoid arthritis 65 99.76 Very High Very High Very High
agonist 2 50.08 Quite High
Inflammation 43 50.00 Quite Low
Arthritis 64 5.00 Very Low Very Low Very Low
cytokine 15 5.00 Very Low Very Low Very Low
methotrexate 4 5.00 Very Low Very Low Very Low
Inflammatory response 3 5.00 Very Low Very Low Very Low
chemokine 2 5.00 Very Low Very Low Very Low
Central nervous system 1 5.00 Very Low Very Low Very Low
Osteoarthritis 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Rheumatoid Arthritis 65 99.76 Very High Very High Very High
Apoptosis 45 99.36 Very High Very High Very High
Medulloblastoma 1 84.00 Quite High
Cancer 12 68.56 Quite High
Death 3 38.40 Quite Low
Necrosis 5 35.64 Quite Low
Arthritis 62 5.00 Very Low Very Low Very Low
INFLAMMATION 17 5.00 Very Low Very Low Very Low
Juvenile Chronic Polyarthritis 3 5.00 Very Low Very Low Very Low
Disease 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In another line of investigation, Audo et al. [128] recently showed that TRAIL-induced apoptosis was inhibited in RA-FLS by the overexpression of the cell survival regulator proteins, p21, X-inhibitor of apotosis protein, MCP-1 and receptor-interacting protein.
Gene_expression (overexpression) of receptor-interacting protein associated with rheumatoid arthritis and apoptosis
1) Confidence 0.04 Published 2009 Journal Mediators of Inflammation Section Body Doc Link PMC2842969 Disease Relevance 0.93 Pain Relevance 0.23

General Comments

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