INT303161

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Context Info
Confidence 0.44
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

intracellular (Trim3, Shank1) cytoplasm (Trim3, Shank1) cell differentiation (Shank1)
plasma membrane (Shank1) protein complex assembly (Shank1)
Trim3 (Rattus norvegicus)
Shank1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
tetrodotoxin 8 5.00 Very Low Very Low Very Low
Glutamate receptor 4 5.00 Very Low Very Low Very Low
imagery 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Angelman Syndrome 2 17.04 Low Low
Syndrome 4 16.04 Low Low
Intellectual Impairment 2 15.68 Low Low
Anxiety Disorder 2 5.00 Very Low Very Low Very Low
Attention Deficit Hyperactivity Disorder 2 5.00 Very Low Very Low Very Low
Cognitive Disorder 2 5.00 Very Low Very Low Very Low
Disease 2 5.00 Very Low Very Low Very Low
Autism 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In addition, proteasomal inhibition by MG-132 did not fully prevent the decrease in Shank1A induced by cotransfection of TRIM3 (data not shown).
Gene_expression (cotransfection) of TRIM3 Positive_regulation (induced) of Shank1A
1) Confidence 0.44 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2844417 Disease Relevance 0 Pain Relevance 0
However, cotransfection with the pooled TRIM3 shRNA constructs led to a highly significant increase in GFP-Shank1A fluorescence (Figure 1A, B).
Gene_expression (cotransfection) of TRIM3 Positive_regulation (increase) of GFP-Shank1A
2) Confidence 0.38 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2844417 Disease Relevance 0 Pain Relevance 0

General Comments

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