INT303655

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Context Info
Confidence 0.16
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 2.19
Pain Relevance 0.41

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (IGF1) extracellular space (IGF1) extracellular region (IGF1)
plasma membrane (SSTR1) cell-cell signaling (SSTR1) signal transducer activity (SSTR1)
IGF1 (Homo sapiens)
SSTR1 (Homo sapiens)
Pain Link Frequency Relevance Heat
Somatostatin 212 96.64 Very High Very High Very High
imagery 12 5.00 Very Low Very Low Very Low
Dopamine 10 5.00 Very Low Very Low Very Low
agonist 6 5.00 Very Low Very Low Very Low
Potency 6 5.00 Very Low Very Low Very Low
Serotonin 4 5.00 Very Low Very Low Very Low
palliative 2 5.00 Very Low Very Low Very Low
dopamine receptor 2 5.00 Very Low Very Low Very Low
metalloproteinase 2 5.00 Very Low Very Low Very Low
antagonist 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 18 99.72 Very High Very High Very High
Cancer 174 98.08 Very High Very High Very High
Adenocarcinoma 6 89.12 High High
Chondrosarcoma 2 80.60 Quite High
Disease 44 67.52 Quite High
Neuroendocrine Cancer 22 50.00 Quite Low
Diarrhoea 20 42.76 Quite Low
Somatostatinoma 8 34.36 Quite Low
Metastasis 28 32.24 Quite Low
Achlorhydria 2 20.64 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Apoptosis [programmed cell death] seems to be induced by two different processes: interaction with the SSTR 3 [53] and inhibition of the Insulin-like Growth Factor I (IGF I), potent antiapoptotic hormone [60].
Positive_regulation (induced) of IGF I Binding (interaction) of SSTR associated with apoptosis
1) Confidence 0.16 Published 2010 Journal J Exp Clin Cancer Res Section Body Doc Link PMC2845555 Disease Relevance 1.10 Pain Relevance 0.20
Apoptosis [programmed cell death] seems to be induced by two different processes: interaction with the SSTR 3 [53] and inhibition of the Insulin-like Growth Factor I (IGF I), potent antiapoptotic hormone [60].
Positive_regulation (induced) of Growth Factor I Binding (interaction) of SSTR associated with apoptosis
2) Confidence 0.14 Published 2010 Journal J Exp Clin Cancer Res Section Body Doc Link PMC2845555 Disease Relevance 1.09 Pain Relevance 0.20

General Comments

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