INT303990

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Context Info
Confidence 0.03
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.35
Pain Relevance 0.10

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular space (Lep, LPL) extracellular region (Lep, LPL) lipid metabolic process (Lep, LPL)
signal transduction (Lep) small molecule metabolic process (LPL) plasma membrane (LPL)
Anatomy Link Frequency
monocytes 3
Lep (Mus musculus)
LPL (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammation 9 77.04 Quite High
antagonist 1 62.68 Quite High
chemokine 3 51.88 Quite High
cytokine 24 49.12 Quite Low
Inflammatory stimuli 2 14.60 Low Low
withdrawal 1 5.00 Very Low Very Low Very Low
endometriosis 1 5.00 Very Low Very Low Very Low
Inflammatory response 1 5.00 Very Low Very Low Very Low
spinal inflammation 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Stress 3 99.24 Very High Very High Very High
Apoptosis 6 86.08 High High
INFLAMMATION 12 77.04 Quite High
Infection 2 24.96 Low Low
Obesity 22 9.52 Low Low
Congenital Anomalies 2 7.76 Low Low
Autoimmune Disease 3 5.00 Very Low Very Low Very Low
Adhesions 2 5.00 Very Low Very Low Very Low
Metabolic Syndrome 2 5.00 Very Low Very Low Very Low
Atherosclerosis 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Thus, leptin was shown to stimulate the oxidative burst in control monocytes [32], and binding of leptin at the macrophage cell surface increases lipoprotein lipase expression through oxidative stress- and PKC-dependent pathways.
leptin Binding (binding) of in monocytes Positive_regulation (increases) of Gene_expression (expression) of lipoprotein lipase in monocytes associated with stress
1) Confidence 0.03 Published 2010 Journal Mediators of Inflammation Section Body Doc Link PMC2846344 Disease Relevance 0.35 Pain Relevance 0.10

General Comments

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