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Context Info
Confidence 0.32
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.86
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

enzyme binding (PTEN, AKT1) cytoplasm (PTEN, AKT1) cytosol (PTEN, AKT1)
cell proliferation (PTEN, AKT1) nucleus (PTEN, AKT1) plasma membrane (PTEN, AKT1)
PTEN (Homo sapiens)
AKT1 (Homo sapiens)
Pain Link Frequency Relevance Heat
metalloproteinase 1 5.00 Very Low Very Low Very Low
pain pelvic 1 5.00 Very Low Very Low Very Low
antagonist 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 16 99.16 Very High Very High Very High
Adenocarcinoma 6 94.04 High High
Endometrial Hyperplasia 4 93.32 High High
Endometrial Cancer 153 90.72 High High
Necrosis 1 80.40 Quite High
Cancer 85 80.04 Quite High
Death 2 79.08 Quite High
Congenital Anomalies 2 67.52 Quite High
Carcinoma 61 61.64 Quite High
Endometroid Carcinoma 21 50.04 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Thus, loss of PTEN function leads to increased levels of phospho-AKT, activation of anti-apoptotic protein, and cell cycle progression [9].
Negative_regulation (loss) of PTEN Positive_regulation (leads) of Positive_regulation (increased) of AKT associated with apoptosis
1) Confidence 0.32 Published 2010 Journal Obstetrics and Gynecology International Section Body Doc Link PMC2846683 Disease Relevance 1.86 Pain Relevance 0

General Comments

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