INT304173

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Context Info
Confidence 0.07
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.18
Pain Relevance 0.06

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (SIGLEC8) cell adhesion (SIGLEC8)
Anatomy Link Frequency
cleavage 1
SIGLEC8 (Homo sapiens)
Pain Link Frequency Relevance Heat
cytokine 24 67.28 Quite High
adenocard 1 44.16 Quite Low
Inflammation 17 5.00 Very Low Very Low Very Low
corticosteroid 5 5.00 Very Low Very Low Very Low
chemokine 4 5.00 Very Low Very Low Very Low
lidocaine 3 5.00 Very Low Very Low Very Low
agonist 2 5.00 Very Low Very Low Very Low
Nerve growth factor 1 5.00 Very Low Very Low Very Low
fibrosis 1 5.00 Very Low Very Low Very Low
withdrawal 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 77 99.40 Very High Very High Very High
Death 22 98.80 Very High Very High Very High
Injury 2 98.04 Very High Very High Very High
Parkinson's Disease 1 95.84 Very High Very High Very High
Asthma 24 5.00 Very Low Very Low Very Low
Disease 20 5.00 Very Low Very Low Very Low
Hypereosinophilic Syndrome 17 5.00 Very Low Very Low Very Low
Eosinophilia 16 5.00 Very Low Very Low Very Low
INFLAMMATION 16 5.00 Very Low Very Low Very Low
Hypersensitivity 15 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
A paradigm has emerged that engagement of Siglec-8 activates the apoptotic pathway involving generation of reactive oxygen species leading to down-stream mitochondrial dysfunction and caspase cleavage before apoptotic death ensues.129
Siglec-8 Binding (engagement) of in cleavage associated with parkinson's disease, apoptosis and death
1) Confidence 0.07 Published 2010 Journal Allergy, Asthma & Immunology Research Section Body Doc Link PMC2846745 Disease Relevance 1.18 Pain Relevance 0.06

General Comments

This test has worked.

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