INT30420

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.67
First Reported 1985
Last Reported 1985
Negated 0
Speculated 1
Reported most in Abstract
Documents 1
Total Number 2
Disease Relevance 0.47
Pain Relevance 0.32

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular space (Crh, Gh1) extracellular region (Crh, Gh1) mitochondrion (Gh1)
response to stress (Crh) cytoplasm (Crh) cytosol (Gh1)
Anatomy Link Frequency
plasma 2
Crh (Rattus norvegicus)
Gh1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
antagonist 2 95.72 Very High Very High Very High
Morphine 2 94.00 High High
Somatostatin 2 75.00 Quite High
Disease Link Frequency Relevance Heat
Stress 6 98.12 Very High Very High Very High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
To examine possible mechanisms through which CRF might mediate the inhibitory action of various stimuli on GH secretion, we have administered antisomatostatin (anti-SS) serum to CRF-injected rats and observed that immunoneutralization of endogenous SS blocked the inhibitory action of CRF on basal plasma GH values.
CRF Spec (might) Positive_regulation (mediate) of Localization (secretion) of GH in plasma
1) Confidence 0.67 Published 1985 Journal Endocrinology Section Abstract Doc Link 2866087 Disease Relevance 0.26 Pain Relevance 0.15
Such observations support the hypothesis that in the rat, endogenous CRF mediates the inhibitory action of noxious stimuli on GH secretion and further suggest that this effect may involve an increased release of endogenous SS.
CRF Positive_regulation (mediates) of Localization (secretion) of GH
2) Confidence 0.45 Published 1985 Journal Endocrinology Section Abstract Doc Link 2866087 Disease Relevance 0.21 Pain Relevance 0.17

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox