INT304505

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Context Info
Confidence 0.01
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.64
Pain Relevance 0.36

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleoplasm (Mapk1) mitochondrion (Mapk1) Golgi apparatus (Mapk1)
protein complex (Mapk1) microtubule organizing center (Mapk1) cytoplasm (Mapk1)
Mapk1 (Rattus norvegicus)
Uts2d (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Rostral ventrolateral medulla 55 99.42 Very High Very High Very High
medulla 11 73.96 Quite High
cerebral cortex 1 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
Inflammatory response 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Pressure Volume 2 Under Development 4 97.08 Very High Very High Very High
Death 62 96.52 Very High Very High Very High
Poisoning 7 16.84 Low Low
Toxicity 2 5.00 Very Low Very Low Very Low
Apoptosis 2 5.00 Very Low Very Low Very Low
Overdose 1 5.00 Very Low Very Low Very Low
Middle Cerebral Artery Infarction 1 5.00 Very Low Very Low Very Low
Heart Rate Under Development 1 5.00 Very Low Very Low Very Low
Critical Illness 1 5.00 Very Low Very Low Very Low
INFLAMMATION 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Pretreatment with microinjection into the bilateral RVLM of ERK activation inhibitor peptide II (1 nmol), which binds specifically to ERK2 to prevent its interaction with MEK [25], exacerbated significantly the hypotension and blunted the augmented power density of the LF component of SAP signals during the pro-life phase (fig. 1), without affecting HR.
ERK2 Binding (binds) of peptide II associated with rostral ventrolateral medulla and pressure volume 2 under development
1) Confidence 0.01 Published 2010 Journal J Biomed Sci Section Body Doc Link PMC2848001 Disease Relevance 0.64 Pain Relevance 0.36

General Comments

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