INT30462

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Context Info
Confidence 0.21
First Reported 1985
Last Reported 1999
Negated 0
Speculated 0
Reported most in Abstract
Documents 2
Total Number 2
Disease Relevance 0.08
Pain Relevance 0.58

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (Gnrhr, Gh1) mitochondrion (Gh1) signal transducer activity (Gnrhr)
cytosol (Gh1) extracellular space (Gh1) signal transduction (Gh1)
Anatomy Link Frequency
Thyroid 2
neurons 2
Gnrhr (Rattus norvegicus)
Gh1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Clonidine 2 92.24 High High
Somatostatin 7 85.60 High High
Morphine 4 75.00 Quite High
Disease Link Frequency Relevance Heat
Decapitation 1 75.76 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Thyroid deprivation induced a 92-fold decrease in GH release from the pituitary; however, the ratio between GH release and GH content was similar in TPTX and normal rats, and human pancreatic growth hormone-releasing factor (GRF) (3 X 10(-8) M) was still able to stimulate residual GH release by hemipituitaries from TPTX rats in a manner similar to that in euthyroid controls (295 and 254% stimulation, respectively).
growth hormone Positive_regulation (stimulate) of Localization (release) of GH in Thyroid
1) Confidence 0.21 Published 1985 Journal Neuroendocrinology Section Abstract Doc Link 2867482 Disease Relevance 0.08 Pain Relevance 0.44
Growth hormone secretagogues (GHS) administered systemically selectively induce growth hormone (GH) release from the pituitary and the expression of Fos protein in arcuate nucleus neurons.
Growth hormone Positive_regulation (induce) of Localization (release) of GH in neurons
2) Confidence 0.19 Published 1999 Journal Neuroendocrinology Section Abstract Doc Link 10461024 Disease Relevance 0 Pain Relevance 0.15

General Comments

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