INT305252

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Context Info
Confidence 0.05
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 1.52
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (Mapk8) signal transduction (Mapk8) mitochondrion (Mapk8)
nucleus (Mapk8) kinase activity (Mapk8) cytoplasm (Mapk8)
Obsty1 (Mus musculus)
Mapk8 (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammation 4 5.00 Very Low Very Low Very Low
cytokine 2 5.00 Very Low Very Low Very Low
ketamine 2 5.00 Very Low Very Low Very Low
depression 2 5.00 Very Low Very Low Very Low
anesthesia 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Obesity 338 100.00 Very High Very High Very High
Aging 248 99.90 Very High Very High Very High
Stress 34 84.32 Quite High
Myocardial Infarction 4 37.44 Quite Low
Body Weight 8 8.24 Low Low
Lifespan 12 5.00 Very Low Very Low Very Low
Diabetes Mellitus 10 5.00 Very Low Very Low Very Low
Insulin Resistance 8 5.00 Very Low Very Low Very Low
Death 8 5.00 Very Low Very Low Very Low
Coronary Heart Disease 6 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Our results showed that aging and obesity independently depressed the phosphorylation
               of Akt and eNOS, stimulated JNK and I?
obesity Positive_regulation (stimulated) of JNK associated with aging and obesity
1) Confidence 0.05 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2852499 Disease Relevance 0.67 Pain Relevance 0
Further scrutiny depicted that
               aging and obesity independently decreased the phosphorylation of Akt and its
               downstream signaling molecule eNOS, stimulated JNK and I?
obesity Positive_regulation (stimulated) of JNK associated with aging and obesity
2) Confidence 0.05 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2852499 Disease Relevance 0.85 Pain Relevance 0

General Comments

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