INT305270

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Context Info
Confidence 0.16
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.28
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular space (Lep, Car2) lyase activity (Car2) intracellular (Lep)
cytoplasm (Car2) lipid metabolic process (Lep) cytosol (Car2)
Anatomy Link Frequency
cardiomyocyte 2
Lep (Mus musculus)
Car2 (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammation 2 5.00 Very Low Very Low Very Low
cytokine 1 5.00 Very Low Very Low Very Low
ketamine 1 5.00 Very Low Very Low Very Low
depression 1 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Hypertension 2 96.52 Very High Very High Very High
Hyperlipidemia 1 95.20 Very High Very High Very High
Insulin Resistance 4 94.56 High High
Hyperphagia 1 93.76 High High
Thrombosis 1 86.60 High High
Increased Venous Pressure Under Development 1 86.00 High High
Hypertrophy 1 85.32 High High
Stress 17 80.40 Quite High
Cardiovascular Disease 1 79.08 Quite High
Disease 1 73.80 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Data from our lab
               revealed that leptin directly suppresses cardiomyocyte contraction and intracellular
                   Ca2+ handling through mechanism(s) related to endothelial
               nitric oxide synthase (eNOS), superoxide (O2?)
               
leptin Negative_regulation (suppresses) of Localization (handling) of Ca2 in cardiomyocyte
1) Confidence 0.16 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2852499 Disease Relevance 1.28 Pain Relevance 0

General Comments

This test has worked.

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