INT306624

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Context Info
Confidence 0.08
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.28
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (Prep, Akt1) cytosol (Prep, Akt1) nucleus (Prep, Akt1)
transport (Akt1) aging (Akt1) enzyme binding (Akt1)
Prep (Mus musculus)
Akt1 (Mus musculus)
Pain Link Frequency Relevance Heat
ketamine 2 5.00 Very Low Very Low Very Low
agonist 2 5.00 Very Low Very Low Very Low
anesthesia 2 5.00 Very Low Very Low Very Low
Immobilon 2 5.00 Very Low Very Low Very Low
imagery 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Stress 120 91.20 High High
Sprains And Strains 4 8.92 Low Low
Targeted Disruption 22 5.00 Very Low Very Low Very Low
Hypertrophy 10 5.00 Very Low Very Low Very Low
Myocardial Infarction 8 5.00 Very Low Very Low Very Low
Hyperplasia 4 5.00 Very Low Very Low Very Low
Coronary Heart Disease 4 5.00 Very Low Very Low Very Low
Frailty 4 5.00 Very Low Very Low Very Low
Cytomegalovirus Infection 4 5.00 Very Low Very Low Very Low
Stroke 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Myostatin overexpression blocked PE-induced activation of Akt (phosphorylation) and NFAT3 (dephosphorylation) (Fig. 7A, B).
PE Positive_regulation (activation) of Akt
1) Confidence 0.08 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2856679 Disease Relevance 0.09 Pain Relevance 0
Since our data suggested that myostatin may interact with the same pathways as PE, we investigated two pathways that were activated by PE in our model: Akt and NFAT3.
PE Positive_regulation (activated) of Akt
2) Confidence 0.08 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2856679 Disease Relevance 0.19 Pain Relevance 0

General Comments

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